Research conducted

Sudden idiopathic deafness is characterized by rapidly progressive hearing loss, almost always affecting only one ear, which occurs within 72 hours.
Most cases are idiopathic, meaning there is no one specific cause.
Symptoms usually appear in the morning, and include fullness in the ear, a feeling of cotton wool in the ear, ear congestion, and sometimes tinnitus (squeaking, ringing) and dizziness.

If you have suddenly, for unknown reasons, stopped hearing in one ear, come to us as soon as possible!
We are looking for people who have experienced sudden, idiopathic (unknown origin), unilateral hearing loss and have not yet received treatment after the onset of symptoms.
If you report to us within 2-5 days of the onset of the first symptoms, you can participate in a clinical trial conducted by top specialists at the MEDINCUS Hearing and Speech Center in Kajetany. All clinical trials at the MEDINCUS Hearing and Speech Center are conducted under the supervision of an independent Bioethics Committee and with the approval of the President of the Office for Registration of Medicinal Products, Medical Devices and Biocidal Products.

Chronic rhinosinusitis (CRS) is one of the most common conditions worldwide, affecting nearly 12% of the adult population.
It is characterized by inflammation of the nose and sinus cavities.
About 20% of patients with PHPD have nasal polyps, i.e.
non-cancerous tissue growths that occur within the nose or paranasal sinuses.
Nasal polyps usually occur on both sides of the nasal cavity; however, it is possible to have them on one side.
Typical symptoms of nasal polyps include nasal blockage, congestion of the nasal mucosa, facial pressure or pain, and weakness or loss of smell, lasting longer than 12 weeks.
They may also be accompanied by leakage of nasal discharge, sneezing, a feeling of discharge running down the back wall of the throat, and breathing through the mouth.

• We are looking for volunteers with severe disease who have had a relapse of symptoms despite standard treatment to participate in clinical trials to better understand the condition and help find new treatments.
• Eligible participants will receive reimbursement for travel expenses and a free health check.
• Each participant will undergo an extensive package of qualifying tests, including diagnostic CT.
• Each participant will receive free concomitant treatment in the form of intranasal corticosteroids.

All clinical trials at the MEDINCUS Hearing and Speech Center are conducted under the supervision of an independent Bioethics Committee and with the approval of the President of the Office for Registration of Medicinal Products, Medical Devices and Biocidal Products.

It is assumed that Meniere’s disease is associated with the expansion of the endolymphatic spaces and the formation of the so-called “hydrocephalus.
hydrocephalus of the vagus.
The causes of hydrops formation are divided into those of a mechanical nature and those of a functional nature.
Mechanical disorders cause obstructions in the flow of endolymph from the site of its formation, the vascular striatum, to the site of absorption, the endolymphatic sac, which consequently leads to the formation of a vestibular hydrocele.
This can lead to a sudden onslaught of dizziness caused by a transient paralysis of the vestibulospinal nerve supply neurons.
Functional disorders, on the other hand, are the result of vasoconstriction, which can result in hypoxia.
Activation of vasomotor disorders can result from physical stimuli such as light, cold, stress, heat, exhaustion, or hormonal disorders.
In a certain group of patients, immunological disorders are considered to be the cause of the condition.
The diagnosis of Meniere’s disease is based on the patient’s history and audiological and vestibular system tests.
The history includes information about sudden repeated attacks of dizziness, usually of a spinning ambient nature, occurring together with tinnitus and unilateral progressive hearing loss.
These symptoms may or may not be preceded by so-called “pre-attack symptoms.
pre-attack symptoms.
Such symptoms include fullness in the ear with increasing noise and hearing impairment.
Seizures can last from a few minutes to several hours.
During a seizure, the patient does not lose consciousness, and falls are rare during a seizure.
After a seizure, the patient feels fatigue, and headaches may also occur.
After a seizure, patients may feel unsteady walking for a period of several days.
Characteristic symptoms of the disease include fluctuating hearing deterioration, episodic dizziness, humming or ringing in the ears usually of the buzzing type.
Symptoms are most severe and troublesome in the early stages; in the later stages, when lesions of the vagus are advanced, acute attacks are replaced by permanent balance disorders with progressive hearing damage.
Hearing tests performed on patients usually confirm the presence of sensorineural hearing disorders.
Hearing deterioration usually occurs after subsequent seizures.
Narożny W., Nyka W.M., Siebert J., Etiopathogenesis, diagnosis and treatment of Meniere’s disease, Selected clinical problems, Via Medica 2007, pp.
152-158 Morawiec-Bajda A.Hydrocephalus of the vagus, [w:] Audiologia Kliniczna, Śliwińska-Kowalska M. (ed.), Mediton, Lodz 2005, p.85-88

Sudden sensorineural hearing loss is defined as sensorineural hearing impairment of cochlear origin.
It appears suddenly, without an identified cause, usually unilaterally, and of varying depth.
Hearing impairment may be accompanied by tinnitus and/or dizziness, as well as a feeling of a blocked ear.
The hearing loss lasts from 12 to 96 hours.
The result of tonal audiometry indicates a loss at least at three adjacent frequencies.
In cases of sudden deafness, differential diagnosis is necessary, based on interdisciplinary cooperation.
When the cause cannot be determined, we speak of an idiopathic form of NNCN.
The main problem in the treatment of sudden deafness is that there is no proven therapeutic method that has reproducible results.
Therefore, our center is engaged in conducting research into new methods of treating this condition.
Compiled from: Marek Rogowski: “Sudden deafness” [w:] Mariola Śliwińska-Kowalska (ed.) “Audiologia Kliniczna”, p.263-269 ,Lodz 2005

Dizziness, can be divided into central and peripheral.
The cause of central disorders can be vascular disorders in the central nervous system, trauma, brain tumors, multiple sclerosis, epilepsy, inflammation within the central nervous system.
Peripheral vertigo, on the other hand, is an area of interest for otolaryngologists.
The most common causes of this type of disorder are benign positional vertigo, so-called “peripheral vertigo”.
(BPPV vertigo), Meniere’s disease, inflammation within the vestibular neuron, and drug-induced vertigo – associated with the ototoxic effects of certain drugs.
For the correct diagnosis of the cause of vertigo, the nature of the dizziness may be crucial, for example, for dizziness of the BPPV type, a short time of their occurrence, counted in seconds, is characteristic, and the attacks are triggered by the so-called “critical head position”.
critical position of the head.
Nystagmus may also occur during seizures.
In Meniere’s disease, vertigo attacks usually last from several minutes to several hours.
Characteristically, dizziness may also be accompanied by noise and a feeling of fullness in the ear and hearing impairment.
Vestibular neuronitis can manifest as very severe dizziness accompanied by vomiting and nystagmus without hearing impairment.
The duration of the discomfort is days or even weeks.
The caloric test in patients with this type of condition is positive.
Damage to the inner ear associated with medications (aminoglycosides, cytostatics, loop diuretics, non-steroidal anti-inflammatory drugs) is not very common, but it should be suspected when taking an ototoxic drug is accompanied by dizziness along with tinnitus and hearing impairment.
Litwin T., Członkowska A., Dizziness in the practice of a neurologist – diagnosis and treatment.
Via Medica, ISSN 1734 -525, pp.78-86

Patients most often describe the accompanying annoying sounds as noise, but they can also be experienced as ringing, screeching, whistling, swishing, bubbling, throbbing, howling, chirping, the rustling of crumpled paper and many other sounds. They can be felt in one or both ears or inside the head. They can also be intermittent or continuous. The nature of tinnitus can vary, as can its intensity and pitch. Tinnitus is a consequence of disorders in various sections of the auditory pathway, the most common disorder being in the inner ear. In the diagnosis of tinnitus, the patient’s history is very important, which can provide information about the probable etiology of tinnitus its nature, onset and course. The second stage in the diagnosis of tinnitus is the otolaryngological examination, which includes: otoscopy with evaluation of the mobility of the tympanic membrane, examination of the nasopharynx and the patency of the auditory trumpet, and evaluation of the existence of possible sites in the head and neck region that may generate the so-called objective noise. The patient should also undergo otoneurological, audiological and imaging diagnostics. Audiological diagnosis should include such tests as audiogram, acoustic otoemission, speech comprehension test, tympanometry, stirrup reflexes. It is also important to perform noise characterization in the patient. Treatment of tinnitus is divided into causal and symptomatic. Tinnitus treatment methods include pharmacotherapy, tinnitus masking, electrostimulation, laser therapy, hyperbaric oxygen chambers, feedback method, psychotherapy, tinnitus habituation, and surgical treatment. Treatment is also being attempted with less conventional methods such as acupuncture and hypnosis. Bartnik G.,Tinnitus – clinically relevant facts,Journal of Otorhinolaryngology, 2003,II,3(7)pp. 57-72 Rogowski M., Tinnitus and Auditory Hypersensitivity, (ed.)Clinical Audiology, Mediton, Lodz 2005, pp.
345-351